Micronutrient therapy
| General | |
| Inflammatory processes in gum tissue are associated with increased free radical formation. Clinical studies have shown a strong decrease in antioxidative capacity in the tissue affected by gingivitis. The reduced antioxidant protection can cause lasting damage to the gum tissue. The microcirculation is also interrupted because the capillary system is impaired. The affected tissue can no longer regenerate, which promotes the formation of cavities. | |
| Increases antioxidant supply - Reduces inflammation | |
| Coenzyme Q10 supports the reduction of inflammation in periodontitis. Coenzyme Q10 also reduces bleeding tendency and promotes firmer gums and a lower tendency to plaque formation. Inflammatory reactions caused by bacterial infestation, such as gingivitis and periodontitis, increase the occurrence of free radicals. In order to prevent uncontrolled reactions and damage to healthy tissue cells, endogenous antioxidant protection systems intervene. Coenzyme Q10 is consumed at a higher rate in these reactions. If the body's own synthesis cannot meet the increased demand, the concentration of coenzymes Q10 in the cells decreases. In addition to increased oxidative damage caused by free radicals, this also leads to energetic depletion of the tissue. Individuals with periodontal disease have been observed with a coenzyme Q10 deficiency of 23 – 63% in affected gingival tissue and 20 – 66% lower Q10 values in blood plasma. Healthy oral bacteria need coenzyme Q10 for their metabolism, but the pathogenic bacteria do not. Supplementation strengthens the natural oral flora and suppresses the pathogenic germs. Alpha-lipoic acid, reduced glutathione, zinc and selenium are further antioxidant micronutrients which are recommended for periodontal diseases with inflammatory processes and can be used preventively in the peri-implant phase. |
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| Fights pathogenic bacteria | |
| Pathogenic bacteria play a central role in inflammatory gum diseases. They attach themselves to teeth and gingival tissue via adhesion processes. The adhesion of different bacterial strains and the subsequent infection leads to the inflammatory reactions in the gum tissue. Cranberry proanthocyanidins (A-type PAC) reduce the adhesiveness of bacteria on gingival tissue. Systemic anti-inflammatory effects of cranberry compounds have been demonstrated in periodontal diseases. Cranberry can suppress the formation of proinflammatory cytokines and chemokines and the local secretion of proteolytic enzymes, thereby slowing the destruction of periodontal tissue. |